Limit or avoid for triple negative breast cancer - foods

Last updated: November 7, 2025

Foods to limit or avoid for triple negative BC

The current webpage is designed to make additional research available concerning foods and beverages or their major bioactive components that have the potential to promote triple negative (ER-/PR-/HER2-) breast cancer. At the bottom of this webpage are links to studies relating to the foods under limit or avoid in Foods to eat & avoid for triple negative breast cancer.

Foods and beverages can promote triple negative breast cancer based on incorporating certain compounds or other factors:

Asparagine (asparagus)
Cadmium (possibly significant levels in dried apricots from Turkey, dark chocolate, free range escargot, flaxseed grown in high cadmium soils, Indian or black mustard, shellfish - especially lobster, oysters & squid i.e., calamari, rice grown in cadmium-contaminated soils, sunflower seeds)
High cholesterol content (butter & ghee, caviar, egg yolk, foie gras, pork liver, squid)
Copper (liver - especially calf & beef, sesame seeds, shellfish - especially oysters & calamari, soy protein isolate, sunflower seeds, tree nuts such as Brazil nuts, cashews, pistachio nuts)
Heme iron (beef, caviar, lamb, liver - beef, chicken, lamb, & pork, shellfish - especially oysters, clams, squid)
High fructose corn syrup (sodas, condiments, juice drinks, other sweetened processed foods)
High omega-6 oils (corn oil, safflower oil, soybean oil, sunflower oil)
Highly salty foods (canned foods, cheese & processed cheese, fast food, frozen dinners, pickled foods, processed meats, soybean paste, added salt, salty condiments, salty snacks)
Soy protein isolate (meat replacement, protein bars, protein shakes)
Sugar and sugar-sweetened foods

Selected breast cancer studies

Hyperglycemia accelerated the metastasis of triple-negative breast cancer via promoting TNFα/Gli-1 axis in endothelial cells Cite
Mei X, Ke C, Gao Z, Yang F, Huang Z, Lu B, et al. Hyperglycemia accelerated the metastasis of triple-negative breast cancer via promoting TNFα/Gli-1 axis in endothelial cells. Biochimica et Biophysica Acta (BBA) - General Subjects. Elsevier BV; 2025;:130875 10.1016/j.bbagen.2025.130875
The role of diet and nutraceuticals in the amelioration of multidrug resistance amongst cancer patients Cite
Banerjee S, Banerjee A, Singhvi G, Taliyan R, Dhar P. The role of diet and nutraceuticals in the amelioration of multidrug resistance amongst cancer patients. Beni-Suef University Journal of Basic and Applied Sciences. Springer Science and Business Media LLC; 2025; 14 10.1186/s43088-025-00706-x
Linoleic acid induces migration and invasion through TLR4 in breast cancer cells Cite
Zaragoza KP, Castillo-Sanchez R, Sanchez-Juarez M, Cortes-Reynosa P, Salazar EP. Linoleic acid induces migration and invasion through TLR4 in breast cancer cells. Tissue and Cell. Elsevier BV; 2025; 96:103009 10.1016/j.tice.2025.103009
High glucose promotes cisplatin chemoresistance in MDA‑MB‑231 breast cancer derived cells through changes in gene expression and multiple signaling pathways Cite
Viedma‑Rodríguez A, Martínez‑Hernández M, Flores‑López L, Velázquez‑Flores M, Esparza‑Garrido R, Prado‑Baeza J, et al. High glucose promotes cisplatin chemoresistance in MDA‑MB‑231 breast cancer derived cells through changes in gene expression and multiple signaling pathways. Biomedical Reports. Spandidos Publications; 2025; 23:1-15 10.3892/br.2025.2064
DNA damage response inhibition is an early event in cadmium-induced breast carcinogenesis Cite
Nail AN, Chavez AV, Bailey AN, Banerjee M, Scott JL, Thomas SD, et al. DNA damage response inhibition is an early event in cadmium-induced breast carcinogenesis. Toxicology and Applied Pharmacology. Elsevier BV; 2025; 502:117439 10.1016/j.taap.2025.117439
CCT196969 inhibits TNBC by targeting the HDAC5/RXRA/ASNS axis to down-regulate asparagine synthesis Cite
Yuan Q, Wang Q, Li J, Yin L, Liu S, Zu X, et al. CCT196969 inhibits TNBC by targeting the HDAC5/RXRA/ASNS axis to down-regulate asparagine synthesis. Journal of Experimental & Clinical Cancer Research. Springer Science and Business Media LLC; 2025; 44 10.1186/s13046-025-03494-5
Glutamate promotes triple-negative breast cancer development through IRE1α/XBP1-mediated macrophage polarization: mechanism insights and therapy Cite
Wang H, Sun J, Jiang Z, Tong Z, Wang C. Glutamate promotes triple-negative breast cancer development through IRE1α/XBP1-mediated macrophage polarization: mechanism insights and therapy. Discover Oncology. Springer Science and Business Media LLC; 2025; 16 10.1007/s12672-025-02790-y
Linoleic acid promotes mammary tumor growth and metastasis to brain and lung in female Balb/cJ mice Cite
Sanchez-Juarez M, Vizcarra-Soto M, Castillo-Sanchez R, Torres-Alamilla P, Cortes-Reynosa P, Acosta-Altamirano G, et al. Linoleic acid promotes mammary tumor growth and metastasis to brain and lung in female Balb/cJ mice. Prostaglandins, Leukotrienes and Essential Fatty Acids. Elsevier BV; 2025;:102687 10.1016/j.plefa.2025.102687
Direct sensing of dietary ω-6 linoleic acid through FABP5-mTORC1 signaling Cite
Koundouros N, Nagiec MJ, Bullen N, Noch EK, Burgos-Barragan G, Li Z, et al. Direct sensing of dietary ω-6 linoleic acid through FABP5-mTORC1 signaling. Science. American Association for the Advancement of Science (AAAS); 2025; 387 10.1126/science.adm9805
The role of the mTOR pathway in breast cancer stem cells (BCSCs): mechanisms and therapeutic potentials Cite
Zhang C, Shu X, Yin C, Hu S, Liu P. The role of the mTOR pathway in breast cancer stem cells (BCSCs): mechanisms and therapeutic potentials. Stem Cell Research & Therapy. Springer Science and Business Media LLC; 2025; 16 10.1186/s13287-025-04218-4
High glucose enhances malignant progression of MDA-MB-231 cells through cumulative effect Cite
Zhang G, Liu Z, Zheng H, Xu Y, Zhang D, Chen Q, et al. High glucose enhances malignant progression of MDA-MB-231 cells through cumulative effect. Toxicology Letters. Elsevier BV; 2025; 403:17-31 10.1016/j.toxlet.2024.11.008
Alcohol cessation and breast cancer risk stratified by hormone receptor status Cite
Terry MB, English DR, Freudenheim JL, Lauby-Secretan B, Gapstur SM. Alcohol cessation and breast cancer risk stratified by hormone receptor status. Breast Cancer Research. Springer Science and Business Media LLC; 2024; 26 10.1186/s13058-024-01937-z
Abstract A020: Elevated extracellular glucose level in tumor microenvironment by hyperglycemia increases glycolysis rate, stiffness, contractility, and motility of breast cancer cells through the cAMP-RhoA-Rock and AMPK-YAP-RhoA axes to promote metastasis Cite
Kim T, Bhat AQ, Choe J, Yoon D. Abstract A020: Elevated extracellular glucose level in tumor microenvironment by hyperglycemia increases glycolysis rate, stiffness, contractility, and motility of breast cancer cells through the cAMP-RhoA-Rock and AMPK-YAP-RhoA axes to promote metastasis. Cancer Research. American Association for Cancer Research (AACR); 2024; 84:A020-A020 10.1158/1538-7445.tumbody-a020
Metabolomics and triple-negative breast cancer: A systematic review Cite
Arenas M, Fargas-Saladié M, Moreno-Solé M, Moyano-Femenia L, Jiménez-Franco A, Canela-Capdevila M, et al. Metabolomics and triple-negative breast cancer: A systematic review. Heliyon. Elsevier BV; 2024; 10:e23628 10.1016/j.heliyon.2023.e23628
Disclosing a metabolic signature of cisplatin resistance in MDA-MB-231 triple-negative breast cancer cells by NMR metabolomics Cite
Carneiro TJ, Carvalho ALMB, Vojtek M, Carmo IF, Marques MPM, Diniz C, et al. Disclosing a metabolic signature of cisplatin resistance in MDA-MB-231 triple-negative breast cancer cells by NMR metabolomics. Cancer Cell International. Springer Science and Business Media LLC; 2023; 23 10.1186/s12935-023-03124-0
Metformin combined with glucose starvation synergistically suppress triple-negative breast cancer by enhanced unfolded protein response Cite
Li Y, Zhang Q, Yang J, He W, Jiang Y, Chen Y, et al. Metformin combined with glucose starvation synergistically suppress triple-negative breast cancer by enhanced unfolded protein response. Biochemical and Biophysical Research Communications. Elsevier BV; 2023; 10.1016/j.bbrc.2023.07.029
Influence of alcohol consumption and alcohol metabolism variants on breast cancer risk among Black women: results from the AMBER consortium Cite
Young KL, Olshan AF, Lunetta K, Graff M, Williams LA, Yao S, et al. Influence of alcohol consumption and alcohol metabolism variants on breast cancer risk among Black women: results from the AMBER consortium. Breast Cancer Research. Springer Science and Business Media LLC; 2023; 25 10.1186/s13058-023-01660-1
Abstract 5954: Dietary sugar promotes breast tumorigenesis partially through upregulating 12 lipoxygenase/PPARd signaling and remodeling breast tumor microenvironment Cite
Chakraborty S, Zuo X, Hedge V, Tran M, Jiang Y, Gagea M, et al. Abstract 5954: Dietary sugar promotes breast tumorigenesis partially through upregulating 12 lipoxygenase/PPARd signaling and remodeling breast tumor microenvironment. Cancer Research. American Association for Cancer Research (AACR); 2022; 82:5954-5954 10.1158/1538-7445.am2022-5954
Mammary adipocytes protect triple-negative breast cancer cells from ferroptosis Cite
Xie Y, Wang B, Zhao Y, Tao Z, Wang Y, Chen G, et al. Mammary adipocytes protect triple-negative breast cancer cells from ferroptosis. Journal of Hematology & Oncology. Springer Science and Business Media LLC; 2022; 15 10.1186/s13045-022-01297-1
Diet impacts triple‐negative breast cancer growth, metastatic potential, chemotherapy responsiveness, and doxorubicin‐mediated cardiac dysfunction Cite
Ramirez MU, Clear KYJ, Cornelius Z, Bawaneh A, Feliz‐Mosquea YR, Wilson AS, et al. Diet impacts triple‐negative breast cancer growth, metastatic potential, chemotherapy responsiveness, and doxorubicin‐mediated cardiac dysfunction. Physiological Reports. Wiley; 2022; 10 10.14814/phy2.15192
The association between the inflammatory potential of diet and the risk of histopathological and molecular subtypes of breast cancer in northwestern Iran: Results from the Breast Cancer Risk and Lifestyle study Cite
Hayati Z, Montazeri V, Shivappa N, Hebert JR, Pirouzpanah S. The association between the inflammatory potential of diet and the risk of histopathological and molecular subtypes of breast cancer in northwestern Iran: Results from the Breast Cancer Risk and Lifestyle study. Cancer. Wiley; 2022; 10.1002/cncr.34183
The influence of metabolic factors and ethnicity on breast cancer risk, treatment and survival: The Oslo ethnic breast cancer study Cite
Lofterød T, Frydenberg H, Veierød MB, Jenum AK, Reitan JB, Wist EA, et al. The influence of metabolic factors and ethnicity on breast cancer risk, treatment and survival: The Oslo ethnic breast cancer study. Acta Oncologica. Informa UK Limited; 2022;:1-9 10.1080/0284186x.2022.2053573
Copper depletion modulates mitochondrial oxidative phosphorylation to impair triple negative breast cancer metastasis Cite
Ramchandani D, Berisa M, Tavarez DA, Li Z, Miele M, Bai Y, et al. Copper depletion modulates mitochondrial oxidative phosphorylation to impair triple negative breast cancer metastasis. Nature Communications. Springer Science and Business Media LLC; 2021; 12 10.1038/s41467-021-27559-z
Novel Mechanisms of Tumor Promotion by the Insulin Receptor Isoform A in Triple-Negative Breast Cancer Cells Cite
Vella V, Giuliano M, La Ferlita A, Pellegrino M, Gaudenzi G, Alaimo S, et al. Novel Mechanisms of Tumor Promotion by the Insulin Receptor Isoform A in Triple-Negative Breast Cancer Cells. Cells. MDPI AG; 2021; 10:3145 10.3390/cells10113145
Connecting copper and cancer: from transition metal signalling to metalloplasia Cite
Ge EJ, Bush AI, Casini A, Cobine PA, Cross JR, DeNicola GM, et al. Connecting copper and cancer: from transition metal signalling to metalloplasia. Nature Reviews Cancer. Springer Science and Business Media LLC; 2021; 10.1038/s41568-021-00417-2
Acute cadmium exposure induces GSDME-mediated pyroptosis in triple-negative breast cancer cells through ROS generation and NLRP3 inflammasome pathway activation Cite
Tang J, Bei M, Zhu J, Xu G, Chen D, Jin X, et al. Acute cadmium exposure induces GSDME-mediated pyroptosis in triple-negative breast cancer cells through ROS generation and NLRP3 inflammasome pathway activation. Environmental Toxicology and Pharmacology. Elsevier BV; 2021; 87:103686 10.1016/j.etap.2021.103686
Hyperglycemic conditions proliferate triple negative breast cancer cells: role of ornithine decarboxylase Cite
Capellen CC, Ortega-Rodas J, Morwitzer MJ, Tofilau HMN, Dunworth M, Casero RA, et al. Hyperglycemic conditions proliferate triple negative breast cancer cells: role of ornithine decarboxylase. Breast Cancer Research and Treatment. Springer Science and Business Media LLC; 2021; 10.1007/s10549-021-06388-0
Continuous Inflammatory Stimulation Leads via Metabolic Plasticity to a Prometastatic Phenotype in Triple-Negative Breast Cancer Cells Cite
Morein D, Rubinstein-Achiasaf L, Brayer H, Dorot O, Pichinuk E, Ben-Yaakov H, et al. Continuous Inflammatory Stimulation Leads via Metabolic Plasticity to a Prometastatic Phenotype in Triple-Negative Breast Cancer Cells. Cells. MDPI AG; 2021; 10:1356 10.3390/cells10061356
Dietary inflammatory potential, oxidative balance score, and risk of breast cancer: findings from the Sister Study Cite
Park YM, Shivappa N, Petimar J, Hodgson ME, Nichols HB, Steck SE, et al. Dietary inflammatory potential, oxidative balance score, and risk of breast cancer: findings from the Sister Study. International Journal of Cancer. Wiley; 2021; 10.1002/ijc.33581
Role of Src/FAK in migration and invasion mediated by extracellular vesicles from MDA-MB-231 cells stimulated with linoleic acid Cite
Ramirez-Ricardo J, Leal-Orta E, Garcia-Hernandez A, Diaz-Aragon R, Cortes-Reynosa P, Thompson-Bonilla R, et al. Role of Src/FAK in migration and invasion mediated by extracellular vesicles from MDA-MB-231 cells stimulated with linoleic acid. Medical Oncology. Springer Science and Business Media LLC; 2021; 38 10.1007/s12032-021-01485-y
Linoleic acid induces secretion of extracellular vesicles from MDA-MB-231 breast cancer cells that mediate cellular processes involved with angiogenesis in HUVECs Cite
Garcia-Hernandez A, Leal-Orta E, Ramirez-Ricardo J, Cortes-Reynosa P, Thompson-Bonilla R, Salazar EP. Linoleic acid induces secretion of extracellular vesicles from MDA-MB-231 breast cancer cells that mediate cellular processes involved with angiogenesis in HUVECs. Prostaglandins & Other Lipid Mediators. Elsevier BV; 2020; 153:106519 10.1016/j.prostaglandins.2020.106519
Mitochondrial copper depletion suppresses triple-negative breast cancer in mice Cite
Cui L, Gouw AM, LaGory EL, Guo S, Attarwala N, Tang Y, et al. Mitochondrial copper depletion suppresses triple-negative breast cancer in mice. Nature Biotechnology. Springer Science and Business Media LLC; 2020; 10.1038/s41587-020-0707-9
Iron promotes breast cancer cell migration via IL-6/JAK2/STAT3 signaling pathways in a paracrine or autocrine IL-6-rich inflammatory environment Cite
Cheng M, Liu P, Xu LX. Iron promotes breast cancer cell migration via IL-6/JAK2/STAT3 signaling pathways in a paracrine or autocrine IL-6-rich inflammatory environment. Journal of Inorganic Biochemistry. Elsevier BV; 2020; 210:111159 10.1016/j.jinorgbio.2020.111159
Abstract A08: Cadmium elicits a differential cytotoxic response in triple-negative breast cancer cells Cite
Godfrey S, Rorie C. Abstract A08: Cadmium elicits a differential cytotoxic response in triple-negative breast cancer cells. Cancer Prevention Research. American Association for Cancer Research (AACR); 2020; 13:A08-A08 10.1158/1940-6215.envcaprev19-a08
SAT-145 Cholesterol Uptake as a Critical Vulnerability in Triple Negative Breast Cancer Cite
O’Neill K. SAT-145 Cholesterol Uptake as a Critical Vulnerability in Triple Negative Breast Cancer. Journal of the Endocrine Society. The Endocrine Society; 2020; 4 10.1210/jendso/bvaa046.1368
Iron intake, oxidative stress‐related genes and breast cancer risk Cite
Chang VC, Cotterchio M, Bondy SJ, Kotsopoulos J. Iron intake, oxidative stress‐related genes and breast cancer risk. International Journal of Cancer. Wiley; 2020; 147:1354-1373 10.1002/ijc.32906
Glucose starvation-induced oxidative stress causes mitochondrial dysfunction and apoptosis via Prohibitin 1 upregulation in human breast cancer cells Cite
Raut GK, Chakrabarti M, Pamarthy D, Bhadra MP. Glucose starvation-induced oxidative stress causes mitochondrial dysfunction and apoptosis via Prohibitin 1 upregulation in human breast cancer cells. Free Radical Biology and Medicine. Elsevier BV; 2019; 145:428-441 10.1016/j.freeradbiomed.2019.09.020
Dietary Fructose Consumption and Triple-Negative Breast Cancer Incidence Cite
Strober JW, Brady MJ. Dietary Fructose Consumption and Triple-Negative Breast Cancer Incidence. Frontiers in Endocrinology. Frontiers Media SA; 2019; 10 10.3389/fendo.2019.00367
Suppression of ferroportin expression by cadmium stimulates proliferation, EMT, and migration in triple-negative breast cancer cells Cite
Shan Z, Wei Z, Shaikh ZA. Suppression of ferroportin expression by cadmium stimulates proliferation, EMT, and migration in triple-negative breast cancer cells. Toxicology and Applied Pharmacology. Elsevier BV; 2018; 356:36-43 10.1016/j.taap.2018.07.017
Impact of pre-diagnostic triglycerides and HDL-cholesterol on breast cancer recurrence and survival by breast cancer subtypes Cite
Lofterød T, Mortensen ES, Nalwoga H, Wilsgaard T, Frydenberg H, Risberg T, et al. Impact of pre-diagnostic triglycerides and HDL-cholesterol on breast cancer recurrence and survival by breast cancer subtypes. BMC Cancer. Springer Science and Business Media LLC; 2018; 18 10.1186/s12885-018-4568-2
High salt induces P-glycoprotein mediated treatment resistance in breast cancer cells through store operated calcium influx Cite
Babaer D, Amara S, Ivy M, Zhao Y, Lammers PE, Titze JM, et al. High salt induces P-glycoprotein mediated treatment resistance in breast cancer cells through store operated calcium influx. Oncotarget. Impact Journals, LLC; 2018; 9:25193-25205 10.18632/oncotarget.25391
Meat intake, methods and degrees of cooking and breast cancer risk in the MCC-Spain study Cite
Boldo E, Castelló A, Aragonés N, Amiano P, Pérez-Gómez B, Castaño-Vinyals G, et al. Meat intake, methods and degrees of cooking and breast cancer risk in the MCC-Spain study. Maturitas. Elsevier BV; 2018; 110:62-70 10.1016/j.maturitas.2018.01.020
Linoleic acid induces an increased response to insulin in MDA‐MB‐231 breast cancer cells Cite
Rodriguez‐Monterrosas C, Diaz‐Aragon R, Cortes‐Reynosa P, Salazar EP. Linoleic acid induces an increased response to insulin in MDA‐MB‐231 breast cancer cells. Journal of Cellular Biochemistry. Wiley; 2018; 119:5413-5425 10.1002/jcb.26694
Metabolic profiles of triple-negative and luminal A breast cancer subtypes in African-American identify key metabolic differences Cite
Tayyari F, Gowda GN, Olopade OF, Berg R, Yang HH, Lee MP, et al. Metabolic profiles of triple-negative and luminal A breast cancer subtypes in African-American identify key metabolic differences. Oncotarget. Impact Journals, LLC; 2018; 9:11677-11690 10.18632/oncotarget.24433
Glutaminase is essential for the growth of triple-negative breast cancer cells with a deregulated glutamine metabolism pathway and its suppression synergizes with mTOR inhibition Cite
Lampa M, Arlt H, He T, Ospina B, Reeves J, Zhang B, et al. Glutaminase is essential for the growth of triple-negative breast cancer cells with a deregulated glutamine metabolism pathway and its suppression synergizes with mTOR inhibition. PLOS ONE. Public Library of Science (PLoS); 2017; 12:e0185092 10.1371/journal.pone.0185092
Cadmium stimulates metastasis-associated phenotype in triple-negative breast cancer cells through integrin and β-catenin signaling Cite
Wei Z, Shaikh ZA. Cadmium stimulates metastasis-associated phenotype in triple-negative breast cancer cells through integrin and β-catenin signaling. Toxicology and Applied Pharmacology. Elsevier BV; 2017; 328:70-80 10.1016/j.taap.2017.05.017
Decreased Iron in Cancer Cells and Their Microenvironment Improves Cytolysis of Breast Cancer Cells by Natural Killer Cells Cite
JIANG X, ELLIOTT RL. Decreased Iron in Cancer Cells and Their Microenvironment Improves Cytolysis of Breast Cancer Cells by Natural Killer Cells. Anticancer Research. International Institute of Anticancer Research; 2017; 37:2297-2305 10.21873/anticanres.11567
Linoleic acid induces migration and invasion through FFAR4- and PI3K-/Akt-dependent pathway in MDA-MB-231 breast cancer cells Cite
Serna-Marquez N, Diaz-Aragon R, Reyes-Uribe E, Cortes-Reynosa P, Salazar EP. Linoleic acid induces migration and invasion through FFAR4- and PI3K-/Akt-dependent pathway in MDA-MB-231 breast cancer cells. Medical Oncology. Springer Science and Business Media LLC; 2017; 34 10.1007/s12032-017-0969-3
Dietary Patterns for Women With Triple-negative Breast Cancer and Dense Breasts Cite
Go Y, Chung M, Park Y. Dietary Patterns for Women With Triple-negative Breast Cancer and Dense Breasts. Nutrition and Cancer. Informa UK Limited; 2016; 68:1281-1288 10.1080/01635581.2016.1225102
Abstract LB-349: Copper depletion as a strategy to affect the tumor microenvironment in breast cancer patients at high risk of relapse and in triple negative preclinical models of breast cancer: Updated results of a phase II study of tetrathiomolybdate (TM) in breast cancer (BC) patients (pts) at high risk for recurrence Cite
Nackos E, Lee S, Willis A, Kornhauser N, Ward M, Cobham M, et al. Abstract LB-349: Copper depletion as a strategy to affect the tumor microenvironment in breast cancer patients at high risk of relapse and in triple negative preclinical models of breast cancer: Updated results of a phase II study of tetrathiomolybdate (TM) in breast cancer (BC) patients (pts) at high risk for recurrence. Tumor Biology. American Association for Cancer Research; 2016; 10.1158/1538-7445.am2016-lb-349
Abstract 4536: Polyunsaturated fatty acids alter the epigenetic landscape in triple-negative breast cancer (TNBC) Cite
Chattin AM, O’Brien M, Pardini RS. Abstract 4536: Polyunsaturated fatty acids alter the epigenetic landscape in triple-negative breast cancer (TNBC). Molecular and Cellular Biology, Genetics. American Association for Cancer Research; 2016; 10.1158/1538-7445.am2016-4536
High glucose and insulin enhance uPA expression, ROS formation and invasiveness in breast cancer-derived cells Cite
Flores-López LA, Martínez-Hernández MG, Viedma-Rodríguez R, Díaz-Flores M, Baiza-Gutman LA. High glucose and insulin enhance uPA expression, ROS formation and invasiveness in breast cancer-derived cells. Cellular Oncology. Springer Science and Business Media LLC; 2016; 39:365-378 10.1007/s13402-016-0282-8
Abstract 3735: Dietary sugar induces tumorigenesis in mammary gland partially through 12 lipoxygenase pathway Cite
Jiang Y, Pan Y, Rhea PR, Tan L, Gagea-Iurascu M, Cohen L, et al. Abstract 3735: Dietary sugar induces tumorigenesis in mammary gland partially through 12 lipoxygenase pathway. Prevention Research. American Association for Cancer Research; 2015; 10.1158/1538-7445.am2015-3735
Arachidonic acid promotes migration and invasion through a PI3K/Akt-dependent pathway in MDA-MB-231 breast cancer cells Cite
Villegas-Comonfort S, Castillo-Sanchez R, Serna-Marquez N, Cortes-Reynosa P, Salazar EP. Arachidonic acid promotes migration and invasion through a PI3K/Akt-dependent pathway in MDA-MB-231 breast cancer cells. Prostaglandins, Leukotrienes and Essential Fatty Acids. Elsevier BV; 2014; 90:169-177 10.1016/j.plefa.2014.01.007
Glucose promotes breast cancer aggression and reduces metformin efficacy Cite
Wahdan-Alaswad R, Fan Z, Edgerton SM, Liu B, Deng X, Arnadottir SS, et al. Glucose promotes breast cancer aggression and reduces metformin efficacy. Cell Cycle. Informa UK Limited; 2013; 12:3759-3769 10.4161/cc.26641
High glucose and insulin differentially modulates proliferation in MCF-7 and MDA-MB-231 cells Cite
Gupta C, Tikoo K. High glucose and insulin differentially modulates proliferation in MCF-7 and MDA-MB-231 cells. Journal of Molecular Endocrinology. Bioscientifica; 2013; 51:119-129 10.1530/jme-13-0062