Tag: BPA

Articles

• Effects of exposure to bisphenol A (BPA) on BC risk and recurrence

News

01/05/19

PSA substitute Bisphenol S may contribute to ER+ BC progression

07/29/17

Maternal butter intake increases BPA tumor-promoting effects in pups

10/15/16

BPA promotes tumor development in mice by binding to ER

11/30/11

Consuming canned soup temporarily increases urinary BPA

09/21/11

Prenatal exposure to BPA induces precancerous changes in offspring

06/18/11

Bisphenol A promotes mammary cancer in mice

05/21/11

Eliminating packaged foods can reduce endocrine disruptor exposure

Studies

• Chemicals Used in Plastic Materials: An Estimate of the Attributable Disease Burden and Costs in the United States Cite
  Trasande L, Krithivasan R, Park K, Obsekov V, Belliveau M. Chemicals Used in Plastic Materials: An Estimate of the Attributable Disease Burden and Costs in the United States. Journal of the Endocrine Society. The Endocrine Society; 2024; 8 10.1210/jendso/bvad163
• Threat or treat: Exposure assessment and risk characterisation of chemical contaminants in soft drinks and chocolate bars in various Polish population age groups Cite
  Marincich L, Protti M, Mandrioli R, Mercolini L, Woźniak Ł. Threat or treat: Exposure assessment and risk characterisation of chemical contaminants in soft drinks and chocolate bars in various Polish population age groups. EFSA Journal. EFSA Journal.; 2023; 21 10.2903/j.efsa.2023.e211011
• SAT426 Long-term Nanomolar Bisphenol A (BPA) Exposure of MCF7 Cells Induces DNA Methylation Changes Cite
  Dela Cruz SEL, Velarde MC, Bagamasbad PD. SAT426 Long-term Nanomolar Bisphenol A (BPA) Exposure of MCF7 Cells Induces DNA Methylation Changes. Journal of the Endocrine Society. The Endocrine Society; 2023; 7 10.1210/jendso/bvad114.1057
• Maternal Bisphenol B Exposure and Mammary Gland Development of Offspring: A Time-Series Analysis Cite
  Ji X, Jiang P, Li Y, Su L, Yue H, Sang N. Maternal Bisphenol B Exposure and Mammary Gland Development of Offspring: A Time-Series Analysis. Environment & Health. American Chemical Society (ACS); 2023; 10.1021/envhealth.3c00085
• Endocrine‐disrupting compounds and metabolomic reprogramming in breast cancer Cite
  Winz C, Zong W, Suh N. Endocrine‐disrupting compounds and metabolomic reprogramming in breast cancer. Journal of Biochemical and Molecular Toxicology. Wiley; 2023; 10.1002/jbt.23506
• A cohort study investigating the role of Bisphenol A in the molecular pathogenesis of breast cancer Cite
  Ishtiaq A, Nasrullah MA, Khan JS, Malik S, Tareen U, Anees M, et al. A cohort study investigating the role of Bisphenol A in the molecular pathogenesis of breast cancer. Journal of Cancer Research and Clinical Oncology. Springer Science and Business Media LLC; 2023; 10.1007/s00432-023-05247-3
• Estrogens and endocrine-disrupting chemicals differentially impact the bioenergetic fluxes of mammary epithelial cells in two- and three-dimensional models Cite
  Lacouture A, Breton Y, Weidmann C, Goulet S, Germain L, Pelletier M, et al. Estrogens and endocrine-disrupting chemicals differentially impact the bioenergetic fluxes of mammary epithelial cells in two- and three-dimensional models. Environment International. Elsevier BV; 2023;:108132 10.1016/j.envint.2023.108132
• Evidence-based identification of breast cancer and associated ovarian and uterus cancer risk components in source waters from high incidence area in the Pearl River Basin, China Cite
  Peng S, Dong S, Gong C, Chen X, Du H, Zhan Y, et al. Evidence-based identification of breast cancer and associated ovarian and uterus cancer risk components in source waters from high incidence area in the Pearl River Basin, China. Science of The Total Environment. Elsevier BV; 2023;:166060 10.1016/j.scitotenv.2023.166060
• Origin, dietary exposure, and toxicity of endocrine-disrupting food chemical contaminants: A comprehensive review Cite
  Peivasteh-roudsari L, Barzegar-bafrouei R, Sharifi KA, Azimisalim S, Karami M, Abedinzadeh S, et al. Origin, dietary exposure, and toxicity of endocrine-disrupting food chemical contaminants: A comprehensive review. Heliyon. Elsevier BV; 2023;:e18140 10.1016/j.heliyon.2023.e18140
• Bisphenol A increases the size of primary mammary tumors and promotes metastasis in a murine model of breast cancer Cite
  Torres-Alamilla P, Castillo-Sanchez R, Cortes-Reynosa P, Gomez-Ortega R, Perez Salazar E. Bisphenol A increases the size of primary mammary tumors and promotes metastasis in a murine model of breast cancer. Molecular and Cellular Endocrinology. Elsevier BV; 2023;:111998 10.1016/j.mce.2023.111998
• Systematic review of the potential carcinogenicity of bisphenol A in humans Cite
  Prueitt RL, Hixon ML, Fan T, Olgun NS, Piatos P, Zhou J, et al. Systematic review of the potential carcinogenicity of bisphenol A in humans. Regulatory Toxicology and Pharmacology. Elsevier BV; 2023;:105414 10.1016/j.yrtph.2023.105414
• Identifying Toxic Consumer Products: A Novel Data Set Reveals Air Emissions of Potent Carcinogens, Reproductive Toxicants, and Developmental Toxicants Cite
  Knox KE, Dodson RE, Rudel RA, Polsky C, Schwarzman MR. Identifying Toxic Consumer Products: A Novel Data Set Reveals Air Emissions of Potent Carcinogens, Reproductive Toxicants, and Developmental Toxicants. Environmental Science & Technology. American Chemical Society (ACS); 2023; 57:7454-7465 10.1021/acs.est.2c07247
• Bisphenol A prevents MCF‐7 breast cell apoptosis via the inhibition of progesterone receptor transactivation Cite
  Ogawa M, Kitamoto J, Takeda T, Terada M. Bisphenol A prevents MCF‐7 breast cell apoptosis via the inhibition of progesterone receptor transactivation. Journal of Biochemical and Molecular Toxicology. Wiley; 2023; 10.1002/jbt.23367
• Food Thermal Labels are a Source of Dietary Exposure to Bisphenol S and Other Color Developers Cite
  XU Z, TIAN L, LIU L, GOODYER CG, HALES BF, BAYEN S. Food Thermal Labels are a Source of Dietary Exposure to Bisphenol S and Other Color Developers. Environmental Science & Technology. American Chemical Society (ACS); 2023; 10.1021/acs.est.2c09390
• Endocrine-active and endocrine-disrupting compounds in food – occurrence, formation and relevance Cite
  Stiefel C, Stintzing F. Endocrine-active and endocrine-disrupting compounds in food – occurrence, formation and relevance. NFS Journal. Elsevier BV; 2023; 10.1016/j.nfs.2023.03.004
• Bisphenol A disruption promotes mammary tumor microenvironment via phenotypic cell polarization and inflammatory response Cite
  Ruiz TFR, Colleta SJ, dos Santos DD, Castro NFC, Cabral ÁS, Calmon MF, et al. Bisphenol A disruption promotes mammary tumor microenvironment via phenotypic cell polarization and inflammatory response. Cell Biology International. Wiley; 2023; 10.1002/cbin.12007
• Bisphenol B and bisphenol AF exposure enhances uterine diseases risks in mouse Cite
  Wu X, Yang X, Tian Y, Xu P, Yue H, Sang N. Bisphenol B and bisphenol AF exposure enhances uterine diseases risks in mouse. Environment International. Elsevier BV; 2023; 173:107858 10.1016/j.envint.2023.107858
• Bisphenol S Increases Cell Number and Stimulates Migration of Endometrial Epithelial Cells Cite
  Benjamin K, Marquez CM, Morta M, Reyes EM, Aragones L, Velarde M. Bisphenol S Increases Cell Number and Stimulates Migration of Endometrial Epithelial Cells. Journal of the ASEAN Federation of Endocrine Societies. Journal of the ASEAN Federation of Endocrine Societies (JAFES); 2022; 0 10.15605/jafes.037.s7
• Exposure to Endocrine-Disrupting Chemicals and Type 2 Diabetes Mellitus in Later Life Cite
  Lin J, Yin R. Exposure to Endocrine-Disrupting Chemicals and Type 2 Diabetes Mellitus in Later Life. Exposure and Health. Springer Science and Business Media LLC; 2022; 10.1007/s12403-022-00486-0
• The Role of Cholesterol in the Interaction of the Lipid Monolayer with the Endocrine Disruptor Bisphenol-A Cite
  Katata VM, Maximino MD, Silva CY, Alessio P. The Role of Cholesterol in the Interaction of the Lipid Monolayer with the Endocrine Disruptor Bisphenol-A. Membranes. MDPI AG; 2022; 12:729 10.3390/membranes12080729
• The effects of endocrine-disrupting chemicals (EDCs) on the epigenome-A short overview Cite
  Buha A, Manic L, Maric D, Tinkov A, Skolny A, Antonijevic B, et al. The effects of endocrine-disrupting chemicals (EDCs) on the epigenome-A short overview. Toxicology Research and Application. SAGE Publications; 2022; 6:239784732211158 10.1177/23978473221115817
• Associations of maternal urinary bisphenol and phthalate concentrations with offspring reproductive development Cite
  Blaauwendraad SM, Jaddoe VW, Santos S, Kannan K, Dohle GR, Trasande L, et al. Associations of maternal urinary bisphenol and phthalate concentrations with offspring reproductive development. Environmental Pollution. Elsevier BV; 2022; 309:119745 10.1016/j.envpol.2022.119745
• Bisphenol A exposures and hormone concentrations in a cohort of women receiving aromatase inhibitor therapy for breast cancer. Cite
  Robien K, Kirstein MN, Pawloski PA, Shapiro AC. Bisphenol A exposures and hormone concentrations in a cohort of women receiving aromatase inhibitor therapy for breast cancer.. Journal of Clinical Oncology. American Society of Clinical Oncology (ASCO); 2022; 40:e12535-e12535 10.1200/jco.2022.40.16_suppl.e12535
• Bisphenol A exposure induces metastatic aggression in low metastatic MCF-7 cells via PGC-1α mediated mitochondrial biogenesis and epithelial-mesenchymal plasticity Cite
  Ansari MI, Bano N, Kainat K, Singh VK, Sharma PK. Bisphenol A exposure induces metastatic aggression in low metastatic MCF-7 cells via PGC-1α mediated mitochondrial biogenesis and epithelial-mesenchymal plasticity. Life Sciences. Elsevier BV; 2022; 302:120649 10.1016/j.lfs.2022.120649
• Role of the Synergistic Interactions of Environmental Pollutants in the Development of Cancer Cite
  Lagunas‐Rangel FA, Linnea‐Niemi JV, Kudłak B, Williams MJ, Jönsson J, Schiöth HB. Role of the Synergistic Interactions of Environmental Pollutants in the Development of Cancer. GeoHealth. American Geophysical Union (AGU); 2022; 6 10.1029/2021gh000552
• Bisphenol A exposure advances puberty onset by changing Kiss1 expression firstly in arcuate nucleus at juvenile period in female rats Cite
  Dong W, He J, Wang J, Sun W, Sun Y, Yu J. Bisphenol A exposure advances puberty onset by changing Kiss1 expression firstly in arcuate nucleus at juvenile period in female rats. Reproductive Toxicology. Elsevier BV; 2022; 10.1016/j.reprotox.2022.04.005
• An overview on the occurrence, fate and human risks of Bisphenol‐A (BPA) present in plastic materials, components and products (MCPs) Cite
  Hahladakis JN, Iacovidou E, Gerassimidou S. An overview on the occurrence, fate and human risks of Bisphenol‐A (BPA) present in plastic materials, components and products (MCPs). Integrated Environmental Assessment and Management. Wiley; 2022; 10.1002/ieam.4611
• Diethylhexyl Phthalate and Bisphenol A Promote Vincristine and Tamoxifen Resistance in Vitro Cite
  Uyar R, Yurdakok-Dikmen B, Turgut Y, Filazi A. Diethylhexyl Phthalate and Bisphenol A Promote Vincristine and Tamoxifen Resistance in Vitro. Chemical Research in Toxicology. American Chemical Society (ACS); 2022; 10.1021/acs.chemrestox.2c00002
• Bisphenol A replacement chemicals, BPF and BPS, induce protumorigenic changes in human mammary gland organoid morphology and proteome Cite
  Winkler J, Liu P, Phong K, Hinrichs JH, Ataii N, Williams K, et al. Bisphenol A replacement chemicals, BPF and BPS, induce protumorigenic changes in human mammary gland organoid morphology and proteome. Proceedings of the National Academy of Sciences. Proceedings of the National Academy of Sciences; 2022; 119 10.1073/pnas.2115308119
• Evaluation of the Cytotoxic Effect of Bisphenol A and Its Analogs in MCF-7 and HSeC Cell Lines in vitro Cite
  İPEK S, İYİGÜNDOĞDU İ, ÜSTÜNDAĞ A, DUYDU Y. Evaluation of the Cytotoxic Effect of Bisphenol A and Its Analogs in MCF-7 and HSeC Cell Lines in vitro. Fabad Journal of Pharmaceutical Sciences. FABAD Farmasotik Bilimler Ankara Dernegi; 2022;:13-22 10.55262/fabadeczacilik.1078759
• The Endocrine Disruptor Compound Bisphenol-A (BPA) Regulates the Intra-Tumoral Immune Microenvironment and Increases Lung Metastasis in an Experimental Model of Breast Cancer Cite
  Palacios-Arreola MI, Moreno-Mendoza NA, Nava-Castro KE, Segovia-Mendoza M, Perez-Torres A, Garay-Canales CA, et al. The Endocrine Disruptor Compound Bisphenol-A (BPA) Regulates the Intra-Tumoral Immune Microenvironment and Increases Lung Metastasis in an Experimental Model of Breast Cancer. International Journal of Molecular Sciences. MDPI AG; 2022; 23:2523 10.3390/ijms23052523
• Can Exposure to Environmental Pollutants Be Associated with Less Effective Chemotherapy in Cancer Patients? Cite
  Lagunas-Rangel FA, Liu W, Schiöth HB. Can Exposure to Environmental Pollutants Be Associated with Less Effective Chemotherapy in Cancer Patients?. International Journal of Environmental Research and Public Health. MDPI AG; 2022; 19:2064 10.3390/ijerph19042064
• Natural and Synthetic Estrogens in Chronic Inflammation and Breast Cancer Cite
  Maharjan CK, Mo J, Wang L, Kim M, Wang S, Borcherding N, et al. Natural and Synthetic Estrogens in Chronic Inflammation and Breast Cancer. Cancers. MDPI AG; 2021; 14:206 10.3390/cancers14010206
• New bisphenol A and bisphenol S analogs: Evaluation of their hERα agonistic and antagonistic activities using the OECD 455 in-vitro assay and molecular modeling Cite
  Durcik M, Hiti L, Tomašič T, Mašič LP. New bisphenol A and bisphenol S analogs: Evaluation of their hERα agonistic and antagonistic activities using the OECD 455 in-vitro assay and molecular modeling. Chemico-Biological Interactions. Elsevier BV; 2022;:109820 10.1016/j.cbi.2022.109820
• New insights on the effects of endocrine-disrupting chemicals on children Cite
  Predieri B, Alves CA, Iughetti L. New insights on the effects of endocrine-disrupting chemicals on children. Jornal de Pediatria. Elsevier BV; 2021; 10.1016/j.jped.2021.11.003
• Dietary Habits and Relationship with the Presence of Main and Trace Elements, Bisphenol A, Tetrabromobisphenol A, and the Lipid, Microbiological and Immunological Profiles of Breast Milk Cite
  Castro I, Arroyo R, Aparicio M, Martínez MÁ, Rovira J, Ares S, et al. Dietary Habits and Relationship with the Presence of Main and Trace Elements, Bisphenol A, Tetrabromobisphenol A, and the Lipid, Microbiological and Immunological Profiles of Breast Milk. Nutrients. MDPI AG; 2021; 13:4346 10.3390/nu13124346
• Endocrine Disruptors–‘Food’ for Thought Cite
  Mukherjee R, Pandya P, Baxi D, Ramachandran AV. Endocrine Disruptors–‘Food’ for Thought. Proceedings of the Zoological Society. Springer Science and Business Media LLC; 2021; 10.1007/s12595-021-00414-1
• Current Aspect of Bisphenol A Toxicology and Its Health Effects Cite
  Khamphaya T, Pouyfung P, Kuraeiad S, Vattanasit U, Yimthiang S. Current Aspect of Bisphenol A Toxicology and Its Health Effects. Trends in Sciences. College of Graduate Studies, Walailak University; 2021; 18:408 10.48048/tis.2021.408
• Regulatory and academic studies to derive reference values for human health: The case of bisphenol S Cite
  Beausoleil C, Le Magueresse-Battistoni B, Viguié C, Babajko S, Canivenc-Lavier M, Chevalier N, et al. Regulatory and academic studies to derive reference values for human health: The case of bisphenol S. Environmental Research. Elsevier BV; 2021;:112233 10.1016/j.envres.2021.112233
• Recent advances in analysis of bisphenols and their derivatives in biological matrices Cite
  Ocaña-Rios I, de Jesús Olmos-Espejel J, Donkor KK. Recent advances in analysis of bisphenols and their derivatives in biological matrices. Analytical and Bioanalytical Chemistry. Springer Science and Business Media LLC; 2021; 10.1007/s00216-021-03668-y
• 4-Methyl-2,4-bis(4-hydroxyphenyl)pent-1-ene (MBP) Targets Estrogen Receptor β, to Evoke the Resistance of Human Breast Cancer MCF-7 Cells to G-1, an Agonist for G Protein-Coupled Estrogen Receptor 1 Cite
  Hirao-Suzuki M, Nagase K, Suemori T, Tsutsumi K, Shigemori E, Tanaka M, et al. 4-Methyl-2,4-bis(4-hydroxyphenyl)pent-1-ene (MBP) Targets Estrogen Receptor β, to Evoke the Resistance of Human Breast Cancer MCF-7 Cells to G-1, an Agonist for G Protein-Coupled Estrogen Receptor 1. Biological and Pharmaceutical Bulletin. Pharmaceutical Society of Japan; 2021; 44:1524-1529 10.1248/bpb.b21-00417
• Bisphenol-A in biological samples of breast cancer mastectomy and mammoplasty patients and correlation with levels measured in urine and tissue Cite
  Keshavarz-Maleki R, Kaviani A, Omranipour R, Gholami M, Khoshayand MR, Ostad SN, et al. Bisphenol-A in biological samples of breast cancer mastectomy and mammoplasty patients and correlation with levels measured in urine and tissue. Scientific Reports. Springer Science and Business Media LLC; 2021; 11 10.1038/s41598-021-97864-6
• Maternal exposure to bisphenol A induces fetal growth restriction via upregulating the expression of estrogen receptors. Cite
  Cao Y, Chen Z, Zhang M, Shi L, Qin S, Lv D, et al. Maternal exposure to bisphenol A induces fetal growth restriction via upregulating the expression of estrogen receptors.. Chemosphere. Elsevier BV; 2022; 287:132244 10.1016/j.chemosphere.2021.132244
• The hazardous threat of Bisphenol A: Toxicity, detection and remediation Cite
  Tarafdar A, Sirohi R, Balakumaran PA, Reshmy R, Madhavan A, Sindhu R, et al. The hazardous threat of Bisphenol A: Toxicity, detection and remediation. Journal of Hazardous Materials. Elsevier BV; 2022; 423:127097 10.1016/j.jhazmat.2021.127097
• Bisphenol A and cancer: a prelude to challenging epidemiology Cite
  Erren TC. Bisphenol A and cancer: a prelude to challenging epidemiology. International Archives of Occupational and Environmental Health. Springer Science and Business Media LLC; 2021; 10.1007/s00420-021-01752-5
• Bisphenol-A exposure and risk of breast and prostate cancer in the Spanish European Prospective Investigation into Cancer and Nutrition study Cite
  Salamanca-Fernández E, Rodríguez-Barranco M, Amiano P, Delfrade J, Chirlaque MD, Colorado S, et al. Bisphenol-A exposure and risk of breast and prostate cancer in the Spanish European Prospective Investigation into Cancer and Nutrition study. Environmental Health. Springer Science and Business Media LLC; 2021; 20 10.1186/s12940-021-00779-y
• Co-exposure to BPA and DEHP enhances susceptibility of mammary tumors via up-regulating Esr1/HDAC6 pathway in female rats Cite
  Zhang X, Cheng C, Zhang G, Xiao M, Li L, Wu S, et al. Co-exposure to BPA and DEHP enhances susceptibility of mammary tumors via up-regulating Esr1/HDAC6 pathway in female rats. Ecotoxicology and Environmental Safety. Elsevier BV; 2021; 221:112453 10.1016/j.ecoenv.2021.112453
• Understanding the Mechanistic Link between Bisphenol A and Cancer Stem Cells: A Cancer Prevention Perspective Cite
  Winz C, Suh N. Understanding the Mechanistic Link between Bisphenol A and Cancer Stem Cells: A Cancer Prevention Perspective. Journal of Cancer Prevention. Korean Society of Cancer Prevention; 2021; 26:18-24 10.15430/jcp.2021.26.1.18
• Bisphenol A and Metabolites in Meat and Meat Products: Occurrence, Toxicity, and Recent Development in Analytical Methods Cite
  Siddique MAb, Harrison SM, Monahan FJ, Cummins E, Brunton NP. Bisphenol A and Metabolites in Meat and Meat Products: Occurrence, Toxicity, and Recent Development in Analytical Methods. Foods. MDPI AG; 2021; 10:714 10.3390/foods10040714
• Breast cancer proliferation and deterioration-associated metabolic heterogeneity changes induced by exposure of bisphenol S, a widespread replacement of bisphenol A Cite
  Zhao C, Yong T, Zhang Y, Xiao Y, Jin Y, Zheng C, et al. Breast cancer proliferation and deterioration-associated metabolic heterogeneity changes induced by exposure of bisphenol S, a widespread replacement of bisphenol A. Journal of Hazardous Materials. Elsevier BV; 2021;:125391 10.1016/j.jhazmat.2021.125391
• Exposure to bisphenol A and breast cancer risk in northern Mexican women Cite
  López-Carrillo L, Mérida-Ortega Á, Gómez-Ruiz H, Hernández-Garciadiego L, Gamboa-Loira B. Exposure to bisphenol A and breast cancer risk in northern Mexican women. International Archives of Occupational and Environmental Health. Springer Science and Business Media LLC; 2021; 10.1007/s00420-020-01590-x
• Isobaric tags for relative and absolute quantitation‑based proteomics analysis of the effect of ginger oil on bisphenol A‑induced breast cancer cell proliferation Cite
  Lei D, Hong T, Li L, Chen L, Luo X, Wu Q, et al. Isobaric tags for relative and absolute quantitation‑based proteomics analysis of the effect of ginger oil on bisphenol A‑induced breast cancer cell proliferation. Oncology Letters. Spandidos Publications; 2020; 21 10.3892/ol.2020.12362
• Metabolomic and Transcriptomic Analysis of MCF-7 Cells Exposed to 23 Chemicals at Human-Relevant Levels: Estimation of Individual Chemical Contribution to Effects Cite
  Liu M, Jia S, Dong T, Zhao F, Xu T, Yang Q, et al. Metabolomic and Transcriptomic Analysis of MCF-7 Cells Exposed to 23 Chemicals at Human-Relevant Levels: Estimation of Individual Chemical Contribution to Effects. Environmental Health Perspectives. Environmental Health Perspectives; 2020; 128:127008 10.1289/ehp6641
• A targeted review on fate, occurrence, risk and health implications of bisphenol analogues Cite
  Catenza CJ, Farooq A, Shubear NS, Donkor KK. A targeted review on fate, occurrence, risk and health implications of bisphenol analogues. Chemosphere. Elsevier BV; 2020; 268:129273 10.1016/j.chemosphere.2020.129273
• Bisphenol A promotes breast cancer cell proliferation by driving miR-381-3p-PTTG1-dependent cell cycle progression Cite
  Deng P, Tan M, Zhou W, Chen C, Xi Y, Gao P, et al. Bisphenol A promotes breast cancer cell proliferation by driving miR-381-3p-PTTG1-dependent cell cycle progression. Chemosphere. Elsevier BV; 2020; 268:129221 10.1016/j.chemosphere.2020.129221
• Bisphenol A and thyroid hormones Cite
  Yuan N, Wang L, Zhang X, Li W. Bisphenol A and thyroid hormones. Medicine. Ovid Technologies (Wolters Kluwer Health); 2020; 99:e23067 10.1097/md.0000000000023067
• Consumer behavior and exposure to parabens, bisphenols, triclosan, dichlorophenols, and benzophenone-3: Results from a crowdsourced biomonitoring study Cite
  Dodson RE, Boronow KE, Susmann H, Udesky JO, Rodgers KM, Weller D, et al. Consumer behavior and exposure to parabens, bisphenols, triclosan, dichlorophenols, and benzophenone-3: Results from a crowdsourced biomonitoring study. International Journal of Hygiene and Environmental Health. Elsevier BV; 2020; 230:113624 10.1016/j.ijheh.2020.113624
• Environmental estrogen exposure converts lipid metabolism in male fish to a female pattern mediated by AMPK and mTOR signaling pathways Cite
  Sun S, Wu J, Lv H, Zhang H, Zhang J, Limbu SM, et al. Environmental estrogen exposure converts lipid metabolism in male fish to a female pattern mediated by AMPK and mTOR signaling pathways. Journal of Hazardous Materials. Elsevier BV; 2020; 394:122537 10.1016/j.jhazmat.2020.122537
• Bisphenol A induces focal adhesions assembly and activation of FAK, Src and ERK2 via GPER in MDA-MB-231 breast cancer cells Cite
  Castillo-Sanchez R, Ramirez-Ricardo J, Martinez-Baeza E, Cortes-Reynosa P, Candanedo-Gonzales F, Gomez R, et al. Bisphenol A induces focal adhesions assembly and activation of FAK, Src and ERK2 via GPER in MDA-MB-231 breast cancer cells. Toxicology in Vitro. Elsevier BV; 2020; 66:104871 10.1016/j.tiv.2020.104871
• Bisphenol-A in the European Prospective Investigation into Cancer and Nutrition cohort in Spain: Levels at recruitment and associated dietary factors Cite
  Salamanca-Fernández E, Rodríguez-Barranco M, Arrebola JP, Vela F, Díaz C, Chirlaque MD, et al. Bisphenol-A in the European Prospective Investigation into Cancer and Nutrition cohort in Spain: Levels at recruitment and associated dietary factors. Environmental Research. Elsevier BV; 2020; 182:109012 10.1016/j.envres.2019.109012
• BPA: have flawed analytical techniques compromised risk assessments? Cite
  Gerona R, vom Saal FS, Hunt PA. BPA: have flawed analytical techniques compromised risk assessments?. The Lancet Diabetes & Endocrinology. Elsevier BV; 2020; 8:11-13 10.1016/s2213-8587(19)30381-x
• Impact of perinatal bisphenol A and 17β estradiol exposure: Comparing hormone receptor response Cite
  Leonel ECR, Campos SGP, Guerra LHA, Bedolo CM, Vilamaior PSL, Calmon MF, et al. Impact of perinatal bisphenol A and 17β estradiol exposure: Comparing hormone receptor response. Ecotoxicology and Environmental Safety. Elsevier BV; 2020; 188:109918 10.1016/j.ecoenv.2019.109918
• Bisphenol S and Bisphenol A disrupt morphogenesis of MCF-12A human mammary epithelial cells Cite
  Atlas E, Dimitrova V. Bisphenol S and Bisphenol A disrupt morphogenesis of MCF-12A human mammary epithelial cells. Scientific Reports. Springer Science and Business Media LLC; 2019; 9 10.1038/s41598-019-52505-x
• Hormone-Like Effects of Bisphenol A on p53 and Estrogen Receptor Alpha in Breast Cancer Cells Cite
  Lloyd V, Morse M, Purakal B, Parker J, Benard P, Crone M, et al. Hormone-Like Effects of Bisphenol A on p53 and Estrogen Receptor Alpha in Breast Cancer Cells. BioResearch Open Access. Mary Ann Liebert Inc; 2019; 8:169-184 10.1089/biores.2018.0048
• Bisphenol A Analogues in Food and Their Hormonal and Obesogenic Effects: A Review Cite
  Andújar , Gálvez-Ontiveros , Zafra-Gómez , Rodrigo , Álvarez-Cubero , Aguilera , et al. Bisphenol A Analogues in Food and Their Hormonal and Obesogenic Effects: A Review. Nutrients. MDPI AG; 2019; 11:2136 10.3390/nu11092136
• Canned food intake and urinary bisphenol a concentrations: a randomized crossover intervention study Cite
  Peng C, Tsai E, Kao T, Lai T, Liang S, Chiu C, et al. Canned food intake and urinary bisphenol a concentrations: a randomized crossover intervention study. Environmental Science and Pollution Research. Springer Science and Business Media LLC; 2019; 26:27999-28009 10.1007/s11356-019-05534-y
• Breast Cancer and Dietary Intake of Endocrine Disruptors: a Review of Recent Literature Cite
  Bowes DA, Halden RU. Breast Cancer and Dietary Intake of Endocrine Disruptors: a Review of Recent Literature. Current Pathobiology Reports. Springer Science and Business Media LLC; 2019; 7:41-46 10.1007/s40139-019-00199-1
• Bisphenol AF promotes estrogen receptor-positive breast cancer cell proliferation through amphiregulin-mediated crosstalk with receptor tyrosine kinase signaling Cite
  Zhao Q, Howard EW, Parris AB, Ma Z, Xing Y, Yang X. Bisphenol AF promotes estrogen receptor-positive breast cancer cell proliferation through amphiregulin-mediated crosstalk with receptor tyrosine kinase signaling. PLOS ONE. Public Library of Science (PLoS); 2019; 14:e0216469 10.1371/journal.pone.0216469
• Evaluation of Prenatal Exposure to Bisphenol Analogues on Development and Long-Term Health of the Mammary Gland in Female Mice Cite
  Tucker DK, Hayes Bouknight S, Brar SS, Kissling GE, Fenton SE. Evaluation of Prenatal Exposure to Bisphenol Analogues on Development and Long-Term Health of the Mammary Gland in Female Mice. Environmental Health Perspectives. Environmental Health Perspectives; 2018; 126:087003 10.1289/ehp3189
• The effects of bisphenol A, benzyl butyl phthalate, and di(2-ethylhexyl) phthalate on estrogen receptor alpha in estrogen receptor-positive cells under hypoxia Cite
  Park C, Lee J, Kong B, Park J, Song H, Choi K, et al. The effects of bisphenol A, benzyl butyl phthalate, and di(2-ethylhexyl) phthalate on estrogen receptor alpha in estrogen receptor-positive cells under hypoxia. Environmental Pollution. Elsevier BV; 2019; 248:774-781 10.1016/j.envpol.2019.02.069
• Bisphenol S induced epigenetic and transcriptional changes in human breast cancer cell line MCF-7 Cite
  Huang W, Zhao C, Zhong H, Zhang S, Xia Y, Cai Z. Bisphenol S induced epigenetic and transcriptional changes in human breast cancer cell line MCF-7. Environmental Pollution. Elsevier BV; 2019; 246:697-703 10.1016/j.envpol.2018.12.084
• The modulatory role of low concentrations of bisphenol A on tamoxifen-induced proliferation and apoptosis in breast cancer cells Cite
  Huang B, Luo N, Wu X, Xu Z, Wang X, Pan X. The modulatory role of low concentrations of bisphenol A on tamoxifen-induced proliferation and apoptosis in breast cancer cells. Environmental Science and Pollution Research. Springer Science and Business Media LLC; 2018; 26:2353-2362 10.1007/s11356-018-3780-6
• Exposure to bisphenol A, chlorophenols, benzophenones, and parabens in relation to reproductive hormones in healthy women: A chemical mixture approach Cite
  Pollack AZ, Mumford SL, Krall JR, Carmichael AE, Sjaarda LA, Perkins NJ, et al. Exposure to bisphenol A, chlorophenols, benzophenones, and parabens in relation to reproductive hormones in healthy women: A chemical mixture approach. Environment International. Elsevier BV; 2018; 120:137-144 10.1016/j.envint.2018.07.028
• Bisphenol-A in breast adipose tissue of breast cancer cases and controls Cite
  Reeves KW, Schneider S, Xue J, Kannan K, Mason H, Johnson M, et al. Bisphenol-A in breast adipose tissue of breast cancer cases and controls. Environmental Research. Elsevier BV; 2018; 167:735-738 10.1016/j.envres.2018.08.033
• Prediction of the combined effects of multiple estrogenic chemicals on MCF-7 human breast cancer cells and a preliminary molecular exploration of the estrogenic proliferative effects and related gene expression Cite
  Yuan S, Huang C, Ji X, Ma M, Rao K, Wang Z. Prediction of the combined effects of multiple estrogenic chemicals on MCF-7 human breast cancer cells and a preliminary molecular exploration of the estrogenic proliferative effects and related gene expression. Ecotoxicology and Environmental Safety. Elsevier BV; 2018; 160:1-9 10.1016/j.ecoenv.2018.05.025
• Bisphenol AF as an activator of human estrogen receptor β1 (ERβ1) in breast cancer cell lines Cite
  Okazaki H, Hirao-Suzuki M, Takeda S, Takemoto Y, Mizunoe R, Haraguchi K, et al. Bisphenol AF as an activator of human estrogen receptor β1 (ERβ1) in breast cancer cell lines. The Journal of Toxicological Sciences. Japanese Society of Toxicology; 2018; 43:321-327 10.2131/jts.43.321
• The BPA-substitute bisphenol S alters the transcription of genes related to endocrine, stress response and biotransformation pathways in the aquatic midge Chironomus riparius (Diptera, Chironomidae) Cite
  Herrero Ó, Aquilino M, Sánchez-Argüello P, Planelló R. The BPA-substitute bisphenol S alters the transcription of genes related to endocrine, stress response and biotransformation pathways in the aquatic midge Chironomus riparius (Diptera, Chironomidae). PLOS ONE. Public Library of Science (PLoS); 2018; 13:e0193387 10.1371/journal.pone.0193387
• Urinary bisphenol A levels in Turkish girls with premature thelarche Cite
  Durmaz E, Asci A, Erkekoglu P, Balcı A, Bircan I, Koçer-Gumusel B. Urinary bisphenol A levels in Turkish girls with premature thelarche. Human & Experimental Toxicology. SAGE Publications; 2018; 37:1007-1016 10.1177/0960327118756720
• Estimation of intake and uptake of bisphenols and triclosan from personal care products by dermal contact Cite
  Lu S, Yu Y, Ren L, Zhang X, Liu G, Yu Y. Estimation of intake and uptake of bisphenols and triclosan from personal care products by dermal contact. Science of The Total Environment. Elsevier BV; 2018; 621:1389-1396 10.1016/j.scitotenv.2017.10.088
• Bisphenol A induces proliferative effects on both breast cancer cells and vascular endothelial cells through a shared GPER-dependent pathway in hypoxia Cite
  Xu F, Wang X, Wu N, He S, Yi W, Xiang S, et al. Bisphenol A induces proliferative effects on both breast cancer cells and vascular endothelial cells through a shared GPER-dependent pathway in hypoxia. Environmental Pollution. Elsevier BV; 2017; 231:1609-1620 10.1016/j.envpol.2017.09.069
• State of the evidence 2017: an update on the connection between breast cancer and the environment Cite
  Gray JM, Rasanayagam S, Engel C, Rizzo J. State of the evidence 2017: an update on the connection between breast cancer and the environment. Environmental Health. Springer Science and Business Media LLC; 2017; 16 10.1186/s12940-017-0287-4
• Inhibitory effects of 3,3′-diindolylmethane on epithelial-mesenchymal transition induced by endocrine disrupting chemicals in cellular and xenograft mouse models of breast cancer Cite
  Lee G, Hwang K, Choi K. Inhibitory effects of 3,3′-diindolylmethane on epithelial-mesenchymal transition induced by endocrine disrupting chemicals in cellular and xenograft mouse models of breast cancer. Food and Chemical Toxicology. Elsevier BV; 2017; 109:284-295 10.1016/j.fct.2017.08.037
• Abstract 4826: Maternal high butter fat intake heightens mammary cancer risk in offsprings gestationally exposed to bisphenol A at environmentally relevant dose Cite
  Leung Y, Govindarajah V, Cheong A, Song D, Zhu X, Ying J, et al. Abstract 4826: Maternal high butter fat intake heightens mammary cancer risk in offsprings gestationally exposed to bisphenol A at environmentally relevant dose. Tumor Biology. American Association for Cancer Research; 2017; 10.1158/1538-7445.am2017-4826
• Bisphenol A exposure assessment from olive oil consumption Cite
  Abou Omar TF, Sukhn C, Fares SA, Abiad MG, Habib RR, Dhaini HR. Bisphenol A exposure assessment from olive oil consumption. Environmental Monitoring and Assessment. Springer Science and Business Media LLC; 2017; 189 10.1007/s10661-017-6048-6
• Altered protein expression of cell cycle- and EMT-associated genes by bisphenol compounds in MCF-7 CV human breast cancer cells via estrogen receptor dependent pathway Cite
  Ayala-Cuellar AP, Hwang K, Choi K. Altered protein expression of cell cycle- and EMT-associated genes by bisphenol compounds in MCF-7 CV human breast cancer cells via estrogen receptor dependent pathway. Endocrine Abstracts. Bioscientifica; 2017; 10.1530/endoabs.49.gp144
• DNA Methylation Targets Influenced by Bisphenol A and/or Genistein Are Associated with Survival Outcomes in Breast Cancer Patients Cite
  Jadhav R, Santucci-Pereira J, Wang Y, Liu J, Nguyen T, Wang J, et al. DNA Methylation Targets Influenced by Bisphenol A and/or Genistein Are Associated with Survival Outcomes in Breast Cancer Patients. Genes. MDPI AG; 2017; 8:144 10.3390/genes8050144
• Bisphenol-A exposure in utero programs a sexually dimorphic estrogenic state of hepatic metabolic gene expression Cite
  Ilagan Y, Mamillapalli R, Goetz TG, Kayani J, Taylor HS. Bisphenol-A exposure in utero programs a sexually dimorphic estrogenic state of hepatic metabolic gene expression. Reproductive Toxicology. Elsevier BV; 2017; 71:84-94 10.1016/j.reprotox.2017.05.001
• Bisphenol A activates EGFR and ERK promoting proliferation, tumor spheroid formation and resistance to EGFR pathway inhibition in estrogen receptor-negative inflammatory breast cancer cells Cite
  Sauer SJ, Tarpley M, Shah I, Save AV, Lyerly H, Patierno SR, et al. Bisphenol A activates EGFR and ERK promoting proliferation, tumor spheroid formation and resistance to EGFR pathway inhibition in estrogen receptor-negative inflammatory breast cancer cells. Carcinogenesis. Oxford University Press (OUP); 2017; 38 10.1093/carcin/bgx003
• Transcriptome profiling reveals bisphenol A alternatives activate estrogen receptor alpha in human breast cancer cells Cite
  Mesnage R, Phedonos A, Arno M, Balu S, Corton JC, Antoniou MN. Transcriptome profiling reveals bisphenol A alternatives activate estrogen receptor alpha in human breast cancer cells. Cold Spring Harbor Laboratory; 2017; 10.1101/112862
• New insights into fetal mammary gland morphogenesis: differential effects of natural and environmental estrogens Cite
  Speroni L, Voutilainen M, Mikkola ML, Klager SA, Schaeberle CM, Sonnenschein C, et al. New insights into fetal mammary gland morphogenesis: differential effects of natural and environmental estrogens. Scientific Reports. Springer Science and Business Media LLC; 2017; 7 10.1038/srep40806
• Perinatal Exposure to Bisphenol A or Diethylstilbestrol Increases the Susceptibility to Develop Mammary Gland Lesions After Estrogen Replacement Therapy in Middle-Aged Rats Cite
  Gomez AL, Delconte MB, Altamirano GA, Vigezzi L, Bosquiazzo VL, Barbisan LF, et al. Perinatal Exposure to Bisphenol A or Diethylstilbestrol Increases the Susceptibility to Develop Mammary Gland Lesions After Estrogen Replacement Therapy in Middle-Aged Rats. Hormones and Cancer. Springer Science and Business Media LLC; 2017; 8:78-89 10.1007/s12672-016-0282-1
• A review of the role of emerging environmental contaminants in the development of breast cancer in women Cite
  Siddique S, Kubwabo C, Harris SA. A review of the role of emerging environmental contaminants in the development of breast cancer in women. Emerging Contaminants. Elsevier BV; 2016; 2:204-219 10.1016/j.emcon.2016.12.003
• Long-term exposure to bisphenol A or benzo(a)pyrene alters the fate of human mammary epithelial stem cells in response to BMP2 and BMP4, by pre-activating BMP signaling Cite
  Clément F, Xu X, Donini CF, Clément A, Omarjee S, Delay E, et al. Long-term exposure to bisphenol A or benzo(a)pyrene alters the fate of human mammary epithelial stem cells in response to BMP2 and BMP4, by pre-activating BMP signaling. Cell Death & Differentiation. Springer Science and Business Media LLC; 2016; 24:155-166 10.1038/cdd.2016.107
• A transgenic mouse model expressing an ERα folding biosensor reveals the effects of Bisphenol A on estrogen receptor signaling Cite
  Sekar TV, Foygel K, Massoud TF, Gambhir SS, Paulmurugan R. A transgenic mouse model expressing an ERα folding biosensor reveals the effects of Bisphenol A on estrogen receptor signaling. Scientific Reports. Springer Science and Business Media LLC; 2016; 6 10.1038/srep34788
• Higher dermal exposure of cashiers to BPA and its association with DNA oxidative damage Cite
  Lv Y, Lu S, Dai Y, Rui C, Wang Y, Zhou Y, et al. Higher dermal exposure of cashiers to BPA and its association with DNA oxidative damage. Environment International. Elsevier BV; 2017; 98:69-74 10.1016/j.envint.2016.10.001
• Environmental estrogen-like endocrine disrupting chemicals and breast cancer Cite
  Morgan M, Deoraj A, Felty Q, Roy D. Environmental estrogen-like endocrine disrupting chemicals and breast cancer. Molecular and Cellular Endocrinology. Elsevier BV; 2017; 457:89-102 10.1016/j.mce.2016.10.003
• Melatonin decreases estrogen receptor binding to estrogen response elements sites on the OCT4 gene in human breast cancer stem cells Cite
  Lopes J, Arnosti D, Trosko JE, Tai M, Zuccari D. Melatonin decreases estrogen receptor binding to estrogen response elements sites on the OCT4 gene in human breast cancer stem cells. Genes & Cancer. Impact Journals, LLC; 2016; 7:209-217 10.18632/genesandcancer.107
• Preferential epigenetic programming of estrogen response after in utero xenoestrogen (bisphenol‐A) exposure Cite
  Jorgensen EM, Alderman MH, Taylor HS. Preferential epigenetic programming of estrogen response after in utero xenoestrogen (bisphenol‐A) exposure. The FASEB Journal. Wiley; 2016; 30:3194-3201 10.1096/fj.201500089r
• Abstract B31: In utero exposure to bisphenol A induces reprogramming of mammary development and tumor risk in MMTV-erbB-2 transgenic mice Cite
  Ma Z, Parris A, Yang X. Abstract B31: In utero exposure to bisphenol A induces reprogramming of mammary development and tumor risk in MMTV-erbB-2 transgenic mice. Other Topics. American Association for Cancer Research; 2016; 10.1158/1557-3125.devbiolca15-b31
• Endocrine disrupting chemical, bisphenol-A, induces breast cancer associated gene HOXB9 expression in vitro and in vivo Cite
  Deb P, Bhan A, Hussain I, Ansari KI, Bobzean SA, Pandita TK, et al. Endocrine disrupting chemical, bisphenol-A, induces breast cancer associated gene HOXB9 expression in vitro and in vivo. Gene. Elsevier BV; 2016; 590:234-243 10.1016/j.gene.2016.05.009
• Bisphenol A Increases the Migration and Invasion of Triple-Negative Breast Cancer Cells via Oestrogen-related Receptor Gamma Cite
  Zhang X, Liu N, Weng S, Wang H. Bisphenol A Increases the Migration and Invasion of Triple-Negative Breast Cancer Cells via Oestrogen-related Receptor Gamma. Basic & Clinical Pharmacology & Toxicology. Wiley; 2016; 119:389-395 10.1111/bcpt.12591
• Quantitative proteomic analyses of mammary organoids reveals distinct signatures after exposure to environmental chemicals Cite
  Williams KE, Lemieux GA, Hassis ME, Olshen AB, Fisher SJ, Werb Z. Quantitative proteomic analyses of mammary organoids reveals distinct signatures after exposure to environmental chemicals. Proceedings of the National Academy of Sciences. Proceedings of the National Academy of Sciences; 2016; 113:E1343-E1351 10.1073/pnas.1600645113
• Effect of bisphenol A on morphology, apoptosis and proliferation in the resting mammary gland of the adult albino rat Cite
  Ibrahim MAA, Elbakry RH, Bayomy NA. Effect of bisphenol A on morphology, apoptosis and proliferation in the resting mammary gland of the adult albino rat. International Journal of Experimental Pathology. Wiley; 2016; 97:27-36 10.1111/iep.12164
• Bisphenol A (BPA) Exposure In Utero Leads to Immunoregulatory Cytokine Dysregulation in the Mouse Mammary Gland: A Potential Mechanism Programming Breast Cancer Risk Cite
  Fischer C, Mamillapalli R, Goetz LG, Jorgenson E, Ilagan Y, Taylor HS. Bisphenol A (BPA) Exposure In Utero Leads to Immunoregulatory Cytokine Dysregulation in the Mouse Mammary Gland: A Potential Mechanism Programming Breast Cancer Risk. Hormones and Cancer. Springer Science and Business Media LLC; 2016; 7:241-251 10.1007/s12672-016-0254-5
• Endocrine disrupting chemicals (EDCs) and female cancer: Informing the patients Cite
  Rachoń D. Endocrine disrupting chemicals (EDCs) and female cancer: Informing the patients. Reviews in Endocrine and Metabolic Disorders. Springer Science and Business Media LLC; 2015; 16:359-364 10.1007/s11154-016-9332-9
• Actions of Bisphenol A and Bisphenol S on the Reproductive Neuroendocrine System During Early Development in Zebrafish Cite
  Qiu W, Zhao Y, Yang M, Farajzadeh M, Pan C, Wayne NL. Actions of Bisphenol A and Bisphenol S on the Reproductive Neuroendocrine System During Early Development in Zebrafish. Endocrinology. The Endocrine Society; 2015; 157:636-647 10.1210/en.2015-1785
• Bisphenol AF stimulates transcription and secretion of C-X-C chemokine ligand 12 to promote proliferation of cultured T47D breast cancer cells Cite
  Li M, Han X, Gao W, Chen F, Shao B. Bisphenol AF stimulates transcription and secretion of C-X-C chemokine ligand 12 to promote proliferation of cultured T47D breast cancer cells. Toxicology. Elsevier BV; 2015; 338:30-36 10.1016/j.tox.2015.09.007
• Low doses of bisphenol A stimulate the proliferation of breast cancer cells via ERK1/2/ERRγ signals Cite
  Song H, Zhang T, Yang P, Li M, Yang Y, Wang Y, et al. Low doses of bisphenol A stimulate the proliferation of breast cancer cells via ERK1/2/ERRγ signals. Toxicology in Vitro. Elsevier BV; 2015; 30:521-528 10.1016/j.tiv.2015.09.009
• Bisphenol A stimulates the epithelial mesenchymal transition of estrogen negative breast cancer cells via FOXA1 signals Cite
  Zhang X, Wang H, Liu N, Ge L. Bisphenol A stimulates the epithelial mesenchymal transition of estrogen negative breast cancer cells via FOXA1 signals. Archives of Biochemistry and Biophysics. Elsevier BV; 2015; 585:10-16 10.1016/j.abb.2015.09.006
• Environmental phenols and pubertal development in girls Cite
  Wolff MS, Teitelbaum SL, McGovern K, Pinney SM, Windham GC, Galvez M, et al. Environmental phenols and pubertal development in girls. Environment International. Elsevier BV; 2015; 84:174-180 10.1016/j.envint.2015.08.008
• Environmental Pesticides and Heavy Metals — Role in Breast Cancer Cite
  Wallace DR. Environmental Pesticides and Heavy Metals — Role in Breast Cancer. Toxicity and Hazard of Agrochemicals. InTech; 2015; 10.5772/60779
• Soy but not bisphenol A (BPA) or the phytoestrogen genistin alters developmental weight gain and food intake in pregnant rats and their offspring Cite
  Cao J, Echelberger R, Liu M, Sluzas E, McCaffrey K, Buckley B, et al. Soy but not bisphenol A (BPA) or the phytoestrogen genistin alters developmental weight gain and food intake in pregnant rats and their offspring. Reproductive Toxicology. Elsevier BV; 2015; 58:282-294 10.1016/j.reprotox.2015.07.077
• Abstract P1-06-06: Meta-analysis reveal novel mechanism of bisphenol A effect on mammary gland Cite
  Gu X, Gao H, Wang D, Bandyopadhyay A, Dong Q, Sun L. Abstract P1-06-06: Meta-analysis reveal novel mechanism of bisphenol A effect on mammary gland. Poster Session Abstracts. American Association for Cancer Research; 2015; 10.1158/1538-7445.sabcs14-p1-06-06
• Bisphenol-A induces expression of HOXC6, an estrogen-regulated homeobox-containing gene associated with breast cancer Cite
  Hussain I, Bhan A, Ansari KI, Deb P, Bobzean SA, Perrotti LI, et al. Bisphenol-A induces expression of HOXC6, an estrogen-regulated homeobox-containing gene associated with breast cancer. Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. Elsevier BV; 2015; 1849:697-708 10.1016/j.bbagrm.2015.02.003
• Bisphenol A Concentration in Breast Milk following Consumption of a Canned Coffee Drink Cite
  Tateoka Y. Bisphenol A Concentration in Breast Milk following Consumption of a Canned Coffee Drink. Journal of Human Lactation. SAGE Publications; 2014; 31:474-478 10.1177/0890334414563732
• Estrogens in the wrong place at the wrong time: Fetal BPA exposure and mammary cancer Cite
  Paulose T, Speroni L, Sonnenschein C, Soto AM. Estrogens in the wrong place at the wrong time: Fetal BPA exposure and mammary cancer. Reproductive Toxicology. Elsevier BV; 2015; 54:58-65 10.1016/j.reprotox.2014.09.012
• In utero and peripubertal exposure to phthalates and BPA in relation to female sexual maturation Cite
  Watkins DJ, Téllez-Rojo MM, Ferguson KK, Lee JM, Solano-Gonzalez M, Blank-Goldenberg C, et al. In utero and peripubertal exposure to phthalates and BPA in relation to female sexual maturation. Environmental Research. Elsevier BV; 2014; 134:233-241 10.1016/j.envres.2014.08.010
• Prenatal Exposure to BPA Alters the Epigenome of the Rat Mammary Gland and Increases the Propensity to Neoplastic Development Cite
  Dhimolea E, Wadia PR, Murray TJ, Settles ML, Treitman JD, Sonnenschein C, et al. Prenatal Exposure to BPA Alters the Epigenome of the Rat Mammary Gland and Increases the Propensity to Neoplastic Development. PLoS ONE. Public Library of Science (PLoS); 2014; 9:e99800 10.1371/journal.pone.0099800
• Gene Expression Profiling Analysis of Bisphenol A-Induced Perturbation in Biological Processes in ER-Negative HEK293 Cells Cite
  Yin R, Gu L, Li M, Jiang C, Cao T, Zhang X. Gene Expression Profiling Analysis of Bisphenol A-Induced Perturbation in Biological Processes in ER-Negative HEK293 Cells. PLoS ONE. Public Library of Science (PLoS); 2014; 9:e98635 10.1371/journal.pone.0098635
• Cell proliferation and apoptosis in rat mammary glands following combinational exposure to bisphenol A and genistein Cite
  Wang J, Jenkins S, Lamartiniere CA. Cell proliferation and apoptosis in rat mammary glands following combinational exposure to bisphenol A and genistein. BMC Cancer. Springer Science and Business Media LLC; 2014; 14 10.1186/1471-2407-14-379
• Prenatal Bisphenol A exposure delays the development of the male rat mammary gland Cite
  Kass L, Durando M, Altamirano GA, Manfroni-Ghibaudo GE, Luque EH, Muñoz-de-Toro M. Prenatal Bisphenol A exposure delays the development of the male rat mammary gland. Reproductive Toxicology. Elsevier BV; 2015; 54:37-46 10.1016/j.reprotox.2014.02.001
• Influence of Bottle-Feeding on Serum Bisphenol A Levels in Infants Cite
  Rhie Y, Nam H, Oh YJ, Kim H, Lee K. Influence of Bottle-Feeding on Serum Bisphenol A Levels in Infants. Journal of Korean Medical Science. Korean Academy of Medical Sciences; 2014; 29:261 10.3346/jkms.2014.29.2.261
• Bisphenol-A and diethylstilbestrol exposure induces the expression of breast cancer associated long noncoding RNA HOTAIR in vitro and in vivo Cite
  Bhan A, Hussain I, Ansari KI, Bobzean SA, Perrotti LI, Mandal SS. Bisphenol-A and diethylstilbestrol exposure induces the expression of breast cancer associated long noncoding RNA HOTAIR in vitro and in vivo. The Journal of Steroid Biochemistry and Molecular Biology. Elsevier BV; 2014; 141:160-170 10.1016/j.jsbmb.2014.02.002
• Coexposure to Phytoestrogens and Bisphenol A Mimics Estrogenic Effects in an Additive Manner Cite
  Katchy A, Pinto C, Jonsson P, Nguyen-Vu T, Pandelova M, Riu A, et al. Coexposure to Phytoestrogens and Bisphenol A Mimics Estrogenic Effects in an Additive Manner. Toxicological Sciences. Oxford University Press (OUP); 2013; 138:21-35 10.1093/toxsci/kft271
• Predictors of urinary bisphenol A and phthalate metabolite concentrations in Mexican children Cite
  Lewis RC, Meeker JD, Peterson KE, Lee JM, Pace GG, Cantoral A, et al. Predictors of urinary bisphenol A and phthalate metabolite concentrations in Mexican children. Chemosphere. Elsevier BV; 2013; 93:2390-2398 10.1016/j.chemosphere.2013.08.038
• Resveratrol regulates the cell viability promoted by 17β-estradiol or bisphenol A via down-regulation of the cross-talk between estrogen receptor α and insulin growth factor-1 receptor in BG-1 ovarian cancer cells Cite
  Kang N, Hwang K, Lee H, Choi D, Choi K. Resveratrol regulates the cell viability promoted by 17β-estradiol or bisphenol A via down-regulation of the cross-talk between estrogen receptor α and insulin growth factor-1 receptor in BG-1 ovarian cancer cells. Food and Chemical Toxicology. Elsevier BV; 2013; 59:373-379 10.1016/j.fct.2013.06.029
• Bisphenol A: A Model Endocrine Disrupting Chemical With a New Potential Mechanism of Action Cite
  Nagel SC, Bromfield JJ. Bisphenol A: A Model Endocrine Disrupting Chemical With a New Potential Mechanism of Action. Endocrinology. The Endocrine Society; 2013; 154:1962-1964 10.1210/en.2013-1370
• Abstract 4874: Bisphenol A exposure negates tamoxifen-mediated chemoprevention in MMTV-erbB2 transgenic mice. Cite
  Yang X, Ma Z, Jiang Y. Abstract 4874: Bisphenol A exposure negates tamoxifen-mediated chemoprevention in MMTV-erbB2 transgenic mice.. Prevention Research. American Association for Cancer Research; 2013; 10.1158/1538-7445.am2013-4874
• Global gene expression analysis reveals pathway differences between teratogenic and non-teratogenic exposure concentrations of bisphenol A and 17β-estradiol in embryonic zebrafish Cite
  Saili KS, Tilton SC, Waters KM, Tanguay RL. Global gene expression analysis reveals pathway differences between teratogenic and non-teratogenic exposure concentrations of bisphenol A and 17β-estradiol in embryonic zebrafish. Reproductive Toxicology. Elsevier BV; 2013; 38:89-101 10.1016/j.reprotox.2013.03.009
• Association between precocious puberty and some endocrine disruptors in human plasma Cite
  Yum T, Lee S, Kim Y. Association between precocious puberty and some endocrine disruptors in human plasma. Journal of Environmental Science and Health, Part A. Informa UK Limited; 2013; 48:912-917 10.1080/10934529.2013.762734
• Endocrine-Disrupting Chemicals (EDCs): In Vitro Mechanism of Estrogenic Activation and Differential Effects on ER Target Genes Cite
  Li Y, Luh CJ, Burns KA, Arao Y, Jiang Z, Teng CT, et al. Endocrine-Disrupting Chemicals (EDCs): In Vitro Mechanism of Estrogenic Activation and Differential Effects on ER Target Genes. Environmental Health Perspectives. Environmental Health Perspectives; 2013; 121:459-466 10.1289/ehp.1205951
• Effect of low dose bisphenol A on the early differentiation of human embryonic stem cells into mammary epithelial cells Cite
  Yang L, Luo L, Ji W, Gong C, Wu D, Huang H, et al. Effect of low dose bisphenol A on the early differentiation of human embryonic stem cells into mammary epithelial cells. Toxicology Letters. Elsevier BV; 2013; 218:187-193 10.1016/j.toxlet.2013.01.026
• Bisphenol-A-induced inactivation of the p53 axis underlying deregulation of proliferation kinetics, and cell death in non-malignant human breast epithelial cells Cite
  Dairkee SH, Luciani-Torres MG, Moore DH, Goodson WH. Bisphenol-A-induced inactivation of the p53 axis underlying deregulation of proliferation kinetics, and cell death in non-malignant human breast epithelial cells. Carcinogenesis. Oxford University Press (OUP); 2012; 34:703-712 10.1093/carcin/bgs379
• A review of dietary and non-dietary exposure to bisphenol-A Cite
  Geens T, Aerts D, Berthot C, Bourguignon J, Goeyens L, Lecomte P, et al. A review of dietary and non-dietary exposure to bisphenol-A. Food and Chemical Toxicology. Elsevier BV; 2012; 50:3725-3740 10.1016/j.fct.2012.07.059
• Occurrence of Eight Bisphenol Analogues in Indoor Dust from the United States and Several Asian Countries: Implications for Human Exposure Cite
  Liao C, Liu F, Guo Y, Moon H, Nakata H, Wu Q, et al. Occurrence of Eight Bisphenol Analogues in Indoor Dust from the United States and Several Asian Countries: Implications for Human Exposure. Environmental Science & Technology. American Chemical Society (ACS); 2012; 46:9138-9145 10.1021/es302004w
• Lifestyle behaviors associated with exposures to endocrine disruptors Cite
  Martina CA, Weiss B, Swan SH. Lifestyle behaviors associated with exposures to endocrine disruptors. NeuroToxicology. Elsevier BV; 2012; 33:1427-1433 10.1016/j.neuro.2012.05.016
• Bisphenol A alters the development of the rhesus monkey mammary gland Cite
  Tharp AP, Maffini MV, Hunt PA, VandeVoort CA, Sonnenschein C, Soto AM. Bisphenol A alters the development of the rhesus monkey mammary gland. Proceedings of the National Academy of Sciences. Proceedings of the National Academy of Sciences; 2012; 109:8190-8195 10.1073/pnas.1120488109
• Migration of phthalates, alkylphenols, bisphenol A and di(2-ethylhexyl)adipate from food packaging Cite
  Fasano E, Bono-Blay F, Cirillo T, Montuori P, Lacorte S. Migration of phthalates, alkylphenols, bisphenol A and di(2-ethylhexyl)adipate from food packaging. Food Control. Elsevier BV; 2012; 27:132-138 10.1016/j.foodcont.2012.03.005
• Assessment of bisphenol A released from reusable plastic, aluminium and stainless steel water bottles Cite
  Cooper JE, Kendig EL, Belcher SM. Assessment of bisphenol A released from reusable plastic, aluminium and stainless steel water bottles. Chemosphere. Elsevier BV; 2011; 85:943-947 10.1016/j.chemosphere.2011.06.060
• The estrogenic potential of salicylate esters and their possible risks in foods and cosmetics Cite
  Zhang Z, Jia C, Hu Y, Sun L, Jiao J, Zhao L, et al. The estrogenic potential of salicylate esters and their possible risks in foods and cosmetics. Toxicology Letters. Elsevier BV; 2012; 209:146-153 10.1016/j.toxlet.2011.12.004
• Perinatal exposure to xenoestrogens impairs mammary gland differentiation and modifies milk composition in Wistar rats Cite
  Kass L, Altamirano GA, Bosquiazzo VL, Luque EH, Muñoz-de-Toro M. Perinatal exposure to xenoestrogens impairs mammary gland differentiation and modifies milk composition in Wistar rats. Reproductive Toxicology. Elsevier BV; 2012; 33:390-400 10.1016/j.reprotox.2012.02.002
• Treatment with bisphenol A and methoxychlor results in the growth of human breast cancer cells and alteration of the expression of cell cycle-related genes, cyclin D1 and p21, via an estrogen receptor-dependent signaling pathway Cite
  Choi K. Treatment with bisphenol A and methoxychlor results in the growth of human breast cancer cells and alteration of the expression of cell cycle-related genes, cyclin D1 and p21, via an estrogen receptor-dependent signaling pathway. International Journal of Molecular Medicine. Spandidos Publications; 2012; 10.3892/ijmm.2012.903
• Embryotoxic and Teratogenic Effects of the Combination of Bisphenol A and Genistein on In Vitro Cultured Postimplantation Rat Embryos Cite
  Xing L, Xu Y, Xiao Y, Shang L, Liu R, Wei X, et al. Embryotoxic and Teratogenic Effects of the Combination of Bisphenol A and Genistein on In Vitro Cultured Postimplantation Rat Embryos. Toxicological Sciences. Oxford University Press (OUP); 2010; 115:577-588 10.1093/toxsci/kfq081
• Canned Soup Consumption and Urinary Bisphenol A: A Randomized Crossover Trial Cite
  Carwile JL. Canned Soup Consumption and Urinary Bisphenol A: A Randomized Crossover Trial. JAMA. American Medical Association (AMA); 2011; 306:2218 10.1001/jama.2011.1721
• Chronic Oral Exposure to Bisphenol A Results in a Nonmonotonic Dose Response in Mammary Carcinogenesis and Metastasis in MMTV-erbB2 Mice Cite
  Jenkins S, Wang J, Eltoum I, Desmond R, Lamartiniere CA. Chronic Oral Exposure to Bisphenol A Results in a Nonmonotonic Dose Response in Mammary Carcinogenesis and Metastasis in MMTV-erbB2 Mice. Environmental Health Perspectives. Environmental Health Perspectives; 2011; 119:1604-1609 10.1289/ehp.1103850
• Perinatal Exposure to Bisphenol A Increases Adult Mammary Gland Progesterone Response and Cell Number Cite
  Ayyanan A, Laribi O, Schuepbach-Mallepell S, Schrick C, Gutierrez M, Tanos T, et al. Perinatal Exposure to Bisphenol A Increases Adult Mammary Gland Progesterone Response and Cell Number. Molecular Endocrinology. The Endocrine Society; 2011; 25:1915-1923 10.1210/me.2011-1129
• Bisphenol A Increases Mammary Cancer Risk in Two Distinct Mouse Models of Breast Cancer Cite
  Weber Lozada K, Keri RA. Bisphenol A Increases Mammary Cancer Risk in Two Distinct Mouse Models of Breast Cancer. Biology of Reproduction. Oxford University Press (OUP); 2011; 85:490-497 10.1095/biolreprod.110.090431
• Food Packaging and Bisphenol A and Bis(2-Ethyhexyl) Phthalate Exposure: Findings from a Dietary Intervention Cite
  Rudel RA, Gray JM, Engel CL, Rawsthorne TW, Dodson RE, Ackerman JM, et al. Food Packaging and Bisphenol A and Bis(2-Ethyhexyl) Phthalate Exposure: Findings from a Dietary Intervention. Environmental Health Perspectives. Environmental Health Perspectives; 2011; 119:914-920 10.1289/ehp.1003170
• Environmental Chemicals in Pregnant Women in the United States: NHANES 2003–2004 Cite
  Woodruff TJ, Zota AR, Schwartz JM. Environmental Chemicals in Pregnant Women in the United States: NHANES 2003–2004. Environmental Health Perspectives. Environmental Health Perspectives; 2011; 119:878-885 10.1289/ehp.1002727